Isoquercitrin Inhibits Lung Cancer Cell Growth Through Triggering Pyroptosis and Ferroptosis.

Isoquercitrin possesses anti-tumor activity in several types of cancers, however, its effects and underlying mechanisms on lung cancer have not been reported. Human lung cancer cell lines as well as normal lung epithelial BEAS-2B cells were treated with isoquercitrin. The influences of isoquercitrin...

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Publicado en:Nutrition & Cancer Vol. 77; no. 2; pp. 299 - 311
Autores principales: Fan, Haiyin, Xu, Pengfei, Zou, Bin, Wang, Huanyuan, Li, Chao, Huang, Jian
Formato: pictorial research tables/charts Journal Article
Publicado: Taylor & Francis Ltd 2025
Acceso en línea:Ver este registro en EBSCOhost
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      pub: Taylor & Francis Ltd
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        10.1080/01635581.2024.2416246
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        atl: Isoquercitrin Inhibits Lung Cancer Cell Growth Through Triggering Pyroptosis and Ferroptosis.
      aug:
        au:
          Fan, Haiyin
          Xu, Pengfei
          Zou, Bin
          Wang, Huanyuan
          Li, Chao
          Huang, Jian
        affil: Department of Thoracic Surgery, Jiangxi Cancer Hospital, Nanchang, Jiangxi Province, China
      sug:
        subj:
          Lung Neoplasms Drug Therapy
          Lung Neoplasms Physiopathology
          Antineoplastic Agents Therapeutic Use
          Antineoplastic Agents Pharmacodynamics
          Quercetin Therapeutic Use
          Quercetin Pharmacodynamics
          Apoptosis Drug Effects
          Treatment Outcomes
          Animal Studies
          Mice
          Models, Biological
          In Vivo Studies
          Cell Proliferation Drug Effects
          Cell Viability Drug Effects
          Cell Line, Tumor Drug Effects
          Intracellular Signaling Peptides and Proteins Metabolism
          In Vitro Studies
          Quercetin Administration and Dosage
          Immunity
          Inflammation
          Reactive Oxygen Species Drug Effects
          Acetylcysteine Metabolism
          Signal Transduction Drug Effects
          Descriptive Statistics
          Data Analysis Software
      ab: Isoquercitrin possesses anti-tumor activity in several types of cancers, however, its effects and underlying mechanisms on lung cancer have not been reported. Human lung cancer cell lines as well as normal lung epithelial BEAS-2B cells were treated with isoquercitrin. The influences of isoquercitrin in vitro were evaluated by determining cell viability, apoptosis, pyroptosis, and ferroptosis. Additionally, A549 tumor-bearing mice were generated to explore the anti-cancer effect of isoquercitrin in vivo. We found that isoquercitrin dose-dependently reduced lung cancer cells' viability, with no toxicity against BEAS-2B cells. Isoquercitrin at 40 μM and 80 μM was used in vitro. Isoquercitrin increased apoptosis, elevated NLRP3 inflammasome activation-mediated pyroptosis, and promoted ferroptosis in lung cancer cells. NLRP3 knockdown and caspase-1 selective inhibitor VX-765 attenuated isoquercitrin-induced pyroptosis and ferroptosis, but not apoptosis. Furthermore, isoquercitrin accelerated ROS generation, while ROS inhibitor N-acetylcysteine abrogated isoquercitrin-induced apoptosis, NLRP3 related-pyroptosis and ferroptosis. In vivo, isoquercitrin (1 mg/kg and 5 mg/kg) inhibited tumor growth, increased apoptosis, NLRP3-related pyroptosis, ferroptosis and ROS generation in tumors. Taken together, isoquercitrin inhibits lung cancer growth by triggering ROS/NLRP3-mediated pyroptosis and ferroptosis, with ROS also directly inducing apoptosis. This suggests that isoquercitrin might be a potential therapeutic agent for lung cancer.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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