Identification of metabolic progression and subtypes in progressive supranuclear palsy by PET molecular imaging.

Introduction: Progressive supranuclear palsy (PSP) is a neurodegenerative disorder with diverse clinical presentations that are linked to tau pathology. Recently, Subtype and Stage Inference (SuStaIn) algorithm, an innovative data-driven method, has been developed to model both the spatial-temporal...

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Publicado en:European Journal of Nuclear Medicine & Molecular Imaging Vol. 52; no. 3; pp. 823 - 836
Autores principales: Wang, Haotian, Wang, Bo, Liao, Yi, Niu, Jiaqi, Chen, Miao, Chen, Xinhui, Dou, Xiaofeng, Yu, Congcong, Zhong, Yan, Wang, Jing, Jin, Nan, Kang, Yixin, Zhang, Hong, Tian, Mei, Luo, Wei
Formato: Journal Article
Publicado: Springer Nature Feb2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Feb2025
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      pub: Springer Nature
      place: New York, New York
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        atl: Identification of metabolic progression and subtypes in progressive supranuclear palsy by PET molecular imaging.
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        au:
          Wang, Haotian
          Wang, Bo
          Liao, Yi
          Niu, Jiaqi
          Chen, Miao
          Chen, Xinhui
          Dou, Xiaofeng
          Yu, Congcong
          Zhong, Yan
          Wang, Jing
          Jin, Nan
          Kang, Yixin
          Zhang, Hong
          Tian, Mei
          Luo, Wei
        affil: https://ror.org/059cjpv64 Department of Neurology, the Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China
      sug:
      ab: Introduction: Progressive supranuclear palsy (PSP) is a neurodegenerative disorder with diverse clinical presentations that are linked to tau pathology. Recently, Subtype and Stage Inference (SuStaIn) algorithm, an innovative data-driven method, has been developed to model both the spatial-temporal progression and subtypes of disease. This study explores PSP progression using 18F-fluorodeoxyglucose (FDG) positron emission tomography (PET) imaging and the SuStaIn algorithm to identify PSP metabolic progression subtypes and understand disease mechanisms. Methods: The study included 72 PSP patients and 70 controls, with an additional 24 PSP patients enrolled as a test set, undergoing FDG-PET, dopamine transporter (DAT) PET, and neuropsychological assessments. The SuStaIn algorithm was employed to analyze the FDG-PET data, identifying progression subtypes and sequences. Results: Two PSP subtypes were identified: the cortical subtype with early prefrontal hypometabolism and the brainstem subtype with initial midbrain alterations. The cortical subtype displayed greater cognitive impairment and DAT reduction than the brainstem subtype. The test set demonstrates the robustness and reproducibility of the findings. Pathway analysis indicated that disruptions in dopaminergic cortico-basal ganglia pathways are crucial for elucidating the mechanisms of cognitive and behavioral impairment in PSP, leading to the two metabolic progression subtypes. Conclusion: This study identified two spatiotemporal progression subtypes of PSP based on FDG-PET imaging, revealing significant differences in metabolic patterns, striatal dopaminergic uptake, and clinical profiles, particularly cognitive impairments. The findings highlight the crucial role of dopaminergic cortico-basal ganglia pathways in PSP pathophysiology, especially in the cortical subtype, providing insights into PSP heterogeneity and potential avenues for personalized treatments.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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