MicroRNA-129-3p Suppresses Tumor Progression and Chemoradioresistance in Head and Neck Squamous Cell Carcinoma.

(1) Background: MicroRNA-129 (miR-129) participates in tumor progression and chemoresistance in various cancer types. In this study, the role of miR-129-3p, the main mature form of miR-129, in tumor progression and chemoradiotherapy resistance in head and neck cancer was evaluated. (2) Methods: RT-P...

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Publicado en:Current Oncology Vol. 32; no. 1; pp. 54 - 69
Autores principales: Yoon, Tae Mi, Kim, Sun-Ae, Jung, Eun Kyung, Kim, Young-Kook, Lee, Kyung-Hwa, Lim, Sang Chul
Formato: Journal Article
Publicado: MDPI Jan2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jan2025
      vid: 32
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      pub: MDPI
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        182443256
        10.3390/curroncol32010054
        182443256
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        atl: MicroRNA-129-3p Suppresses Tumor Progression and Chemoradioresistance in Head and Neck Squamous Cell Carcinoma.
      aug:
        au:
          Yoon, Tae Mi
          Kim, Sun-Ae
          Jung, Eun Kyung
          Kim, Young-Kook
          Lee, Kyung-Hwa
          Lim, Sang Chul
        affil: Departments of Otorhinolaryngology-Head and Neck Surgery, Chonnam National University Medical School, Hwasun Hospital, Hwasun 58128, Jeonnam, Republic of Korea
      sug:
      ab: (1) Background: MicroRNA-129 (miR-129) participates in tumor progression and chemoresistance in various cancer types. In this study, the role of miR-129-3p, the main mature form of miR-129, in tumor progression and chemoradiotherapy resistance in head and neck cancer was evaluated. (2) Methods: RT-PCR, western blotting, cell proliferation assays, cell apoptosis assays, and cell invasion and migration assays were used. (3) Results: In this study, the miR-129-3p overexpression suppressed the proliferation, invasion, and migration of SNU1041, SCC15, and SCC25 human HNSCC cell lines. Additionally, it induced apoptosis and enhanced radiation-/cisplatin-induced apoptosis of SNU1041, SCC15, and SCC25 cells. Therefore, miR-129-3p could suppress tumor progression and enhance chemoradiosensitivity in human HNSCC. Furthermore, miR-129-3p was downregulated in fresh tumor tissues from patients with HNSCC compared with that in the adjacent normal mucosa. In a nude mouse xenograft model with SNU15 cells, the miR-129-3p overexpression significantly decreased tumor growth, as measured by tumor weight and volume. (4) Conclusions: Our study provides evidence that miR-129-3p suppresses tumor progression and chemoradioresistance in HNSCC. This finding may serve as a basis for developing miR-129-3p-based therapeutic strategies.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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