Wenjing Zhitong Decoction Alleviates Primary Dysmenorrhea Mediated by Pain Sensitization Via the ERα‐BDNF Signaling Pathway.

Background: Wen‐Jing‐Zhi‐Tong decoction (WJZTD) is a traditional Chinese medicine herbal decoction that has demonstrated clinical efficacy in treating primary dysmenorrhea (PDM) over decades. However, the underlying therapeutic mechanism of WJZTD requires further clarification. Objective: This study...

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Publicado en:Journal of Clinical Pharmacy & Therapeutics Vol. 2025; pp. 1 - 19
Autores principales: Qin, Yajie, Yang, Xiaotian, Tang, Xingran, Zhao, Huijin, Zhou, Huifang, Chauhan, Anjali
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 5/14/2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 5/14/2025
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      pub: Wiley-Blackwell
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        atl: Wenjing Zhitong Decoction Alleviates Primary Dysmenorrhea Mediated by Pain Sensitization Via the ERα‐BDNF Signaling Pathway.
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        au:
          Qin, Yajie
          Yang, Xiaotian
          Tang, Xingran
          Zhao, Huijin
          Zhou, Huifang
          Chauhan, Anjali
        affil: The First Clinical Medical College,, Nanjing University of Chinese Medicine,, Nanjing, Jiangsu, China, njucm.edu.cn
      sug:
        subj:
          Dysmenorrhea Drug Therapy
          Medicine, Chinese Traditional
          Drugs, Chinese Herbal Therapeutic Use
          Drugs, Chinese Herbal Pharmacodynamics
          Drug Efficacy Evaluation
          Metabolomics
          Receptors, Estrogen
          Brain-Derived Neurotrophic Factor
          Signal Transduction
          Animal Studies
          Rats
          Funding Source
          Network Pharmacology
          In Vitro Studies
          In Vivo Studies
          Prostaglandins Blood
          Fluorescent Antibody Technique
          Blotting, Western
          Ganglia, Sensory
      ab: Background: Wen‐Jing‐Zhi‐Tong decoction (WJZTD) is a traditional Chinese medicine herbal decoction that has demonstrated clinical efficacy in treating primary dysmenorrhea (PDM) over decades. However, the underlying therapeutic mechanism of WJZTD requires further clarification. Objective: This study aims to explore and elucidate the therapeutic mechanism of WJZTD in treating PDM through metabolomics, integrated network pharmacology, as well as in vitro and in vivo experiments. Methods: A PDM rat model was established by estradiol benzoate and oxytocin for 3 consecutive estrous cycles, and then WJZTD treatment was administered for 3 consecutive estrous cycles. To evaluate the therapeutic effect of WJZTD on PDM, behavioral tests, histological analysis, and evaluation of serum prostaglandins (PGs) were performed. Moreover, immunofluorescence and Western blot analysis were carried out in dorsal root ganglion (DRG) tissues to explore the role of WJZTD in reducing pain sensitivity. Metabolomics and integrated pharmacology were utilized to identify potential bioactive targets, which were then validated through in vitro experiments. Results: WJZTD significantly reduces pain sensitivity in PDM rats, treating the condition effectively by decreasing brain‐derived neurotrophic factor (BDNF) protein expression, c‐Fos‐positive neurons in DRG, and serum prostaglandin PGF2α (PGF2α) levels. A nontargeted metabolic analysis identified 11 differential metabolites, suggesting ESR1 (estrogen receptor α, ERα) as a potential target. In vitro experiments confirmed WJZTD's efficacy in treating PDM through regulating the ERα/BDNF signaling pathway. Conclusion: WJZTD decreases the excitability of DRG neurons in rats with PDM through the ERα/BDNF pathway, which enhances pain sensitization and contributes to reducing dysmenorrhea.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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