Head-to-Head comparison of [18F]FDG, [18F]FMZ, and [18F]SynVesT-1 positron emission tomography imaging in patients with drug-resistant epilepsy.

Purpose: The loss of synaptic vesicle glycoprotein 2 A (SV2A) can lead to dysfunction of GABAergic neurons, but a direct comparison of SV2A and GABAA receptor densities in humans has not been assessed. This study evaluated SV2A and GABAA receptor abnormalities in patients with drug-resistant epileps...

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Published in:European Journal of Nuclear Medicine & Molecular Imaging Vol. 52; no. 7; pp. 2258 - 2267
Main Authors: Li, Guanglei, Lin, Zengping, Bao, Weiqi, Jiang, Shize, Wang, Jie, Huang, Qi, Yang, Yang, He, Juanjuan, Huang, Yiyun, Guan, Yihui, Hu, Jie, Xie, Fang
Format: Journal Article
Published: Springer Nature Jun2025
Online Access:View this record in EBSCOhost
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      dt: Jun2025
      vid: 52
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      pub: Springer Nature
      place: New York, New York
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        182560369
        10.1007/s00259-025-07111-7
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        atl: Head-to-Head comparison of [18F]FDG, [18F]FMZ, and [18F]SynVesT-1 positron emission tomography imaging in patients with drug-resistant epilepsy.
      aug:
        au:
          Li, Guanglei
          Lin, Zengping
          Bao, Weiqi
          Jiang, Shize
          Wang, Jie
          Huang, Qi
          Yang, Yang
          He, Juanjuan
          Huang, Yiyun
          Guan, Yihui
          Hu, Jie
          Xie, Fang
        affil: https://ror.org/013q1eq08 Department of Nuclear Medicine & PET Center, Huashan Hospital, Fudan University, Shanghai, China
      sug:
      ab: Purpose: The loss of synaptic vesicle glycoprotein 2 A (SV2A) can lead to dysfunction of GABAergic neurons, but a direct comparison of SV2A and GABAA receptor densities in humans has not been assessed. This study evaluated SV2A and GABAA receptor abnormalities in patients with drug-resistant epilepsy (DRE) and compared the patterns to glucose hypometabolism. Methods: Eleven patients with DRE were retrospectively recruited and underwent PET imaging with [18F]fluorodeoxyglucose ([18F]FDG), [18F]Flumazenil (FMZ), and [18F]SynVesT-1. Visual assessments counted abnormal metabolic brain regions based on the Anatomical Automatic Labeling (AAL) atlas, while voxel-level analyses delineated the abnormal metabolic distributions. The relationship between hypo-metabolic distributions and the age of epilepsy onset was analyzed. Results: The hypometabolic regions in [18F]FDG PET, identified in the AAL atlas, was significantly broader than in [18F]FMZ (p = 0.0005) and [18F]SynVesT-1 (p = 0.0010) PET, with no statistical difference observed between [18F]FMZ and [18F]SynVesT-1 PET (p > 0.05). The voxel number in [18F]FDG PET was significantly higher than that of the [18F]FMZ and [18F]SynVesT-1 PET in both hypo-intensity area and severe hypo-intensity area. The ratio of the voxel number between these two area was higher for [18F]SynVesT-1 PET compared to [18F]FDG PET (p = 0.0195) and [18F]FMZ PET (p = 0.0237), and positively correlated with the age of epilepsy onset (r = 0.7397, p = 0.0145). Conclusions: [18F]FMZ and [18F]SynVesT-1 PET images revealed a more restricted pattern of reduced uptake compared to [18F]FDG PET in DRE patients. The age of epilepsy onset correlated with a reduction in [18F]SynVesT-1 uptake but not in [18F]FMZ or [18F]FDG uptake.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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