Children With Fragile X Syndrome Display a Switch Towards Fast Fibres in Their Recruitment Strategy During Gait.

Background: Fragile X Syndrome (FXS) is a genetic disorder caused by the lack of FMRP, a crucial protein for brain development and function. FMR1 mutations are categorized into premutation and full mutation (FXSFull), with somatic mosaicism (FXSMos) modulating the FXS phenotype. Recent studies ident...

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Published in:Journal of Intellectual Disability Research Vol. 69; no. 7; pp. 582 - 592
Main Authors: Spolaor, Fabiola, Beghetti, Federica, Piatkowska, Weronika, Guiotto, Annamaria, Polli, Roberta, Bettella, Elisa, Liani, Valentina, di Giorgio, Elisa, Sawacha, Zimi
Format: equations & formulas research tables/charts Journal Article
Published: Wiley-Blackwell Jul2025
Online Access:View this record in EBSCOhost
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      dt: Jul2025
      vid: 69
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1111/jir.13238
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        atl: Children With Fragile X Syndrome Display a Switch Towards Fast Fibres in Their Recruitment Strategy During Gait.
      aug:
        au:
          Spolaor, Fabiola
          Beghetti, Federica
          Piatkowska, Weronika
          Guiotto, Annamaria
          Polli, Roberta
          Bettella, Elisa
          Liani, Valentina
          di Giorgio, Elisa
          Sawacha, Zimi
        affil: Department of Women's and Children's Health, University of Padua, Padua, Italy
      sug:
        subj:
          Fragile X Syndrome
          Muscle Fibers Physiology
          Neural Transmission
          Gait Analysis
          Electromyography Methods
          Human
          Nerve Tissue Proteins
          Mutation
          Videorecording
          Gastrocnemius Muscle
          Tibialis Anterior Muscle
          Rectus Femoris Muscles
          Biceps Brachii Muscles
          Funding Source
          Musculoskeletal System Pathology
          Walking Speed
          Exercise Tolerance
      ab: Background: Fragile X Syndrome (FXS) is a genetic disorder caused by the lack of FMRP, a crucial protein for brain development and function. FMR1 mutations are categorized into premutation and full mutation (FXSFull), with somatic mosaicism (FXSMos) modulating the FXS phenotype. Recent studies identified muscle activity alterations during gait in FXS children. This study aims to explore the relationship between these muscle activity changes and motor fibre recruitment strategies during gait in FXS children. Methods: Fifty‐four FXS children and fourteen healthy controls participated in the study. Gait trials at self‐selected speeds were recorded using four synchronized cameras and a surface electromyography system that captured bilateral activity of Gastrocnemius lateralis, Tibialis anterior, Rectus and Biceps femoris muscles. The continuous wavelet transform, using the 'bump' mother wavelet, provided the percentage distribution of signal energy across nine frequency bands (50‐Hz increments within a 450‐ to 10‐Hz spectrum) and the Instantaneous MeaN Frequency (IMNF) time‐frequency distribution. Results: Results indicated that both FXSFull and FXSMos children exhibit a distinct fibre recruitment strategy compared to controls, with a higher percentage of total energy and elevated IMNF (p < 0.05). Conclusions: This increased reliance on fast‐twitch fibres may contribute to the observed fatigability and exercise intolerance in FXS children.
      pubtype: Academic Journal
      doctype:
        equations & formulas
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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