Moist‐Exposed Burn Ointment Promotes Angiogenesis During Full‐Thickness Wound Healing by Activating the SDF‐1/CXCR4 Axis in a Hyperglycaemic Rat Model.

The mechanism by which moist‐exposed burn ointment (MEBO) promotes wound healing in diabetic foot ulcers (DFUs) remains unclear. To elucidate this mechanism, we investigated the role of stromal cell‐derived factor‐1 (SDF‐1) in MEBO's ability to enhance ulcer healing and neovascularisation using a fu...

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Publicado en:Wound Repair & Regeneration Vol. 33; no. 4; pp. 1 - 15
Autores principales: Zhang, Naili, Fu, Li, Xu, Jiayuan, Gu, Chengxu, Li, Hongxing, Wang, Dong, Liu, Tongshen, Shi, Xueying, Xu, Ning, Qiu, Jun, Ma, Lina
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell Jul/Aug2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul/Aug2025
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      place: Malden, Massachusetts
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        atl: Moist‐Exposed Burn Ointment Promotes Angiogenesis During Full‐Thickness Wound Healing by Activating the SDF‐1/CXCR4 Axis in a Hyperglycaemic Rat Model.
      aug:
        au:
          Zhang, Naili
          Fu, Li
          Xu, Jiayuan
          Gu, Chengxu
          Li, Hongxing
          Wang, Dong
          Liu, Tongshen
          Shi, Xueying
          Xu, Ning
          Qiu, Jun
          Ma, Lina
        affil: Department of Human Anatomy, School of Basic Medical Science, Binzhou Medical University, Yantai Shandong,, China
      sug:
        subj:
          Burns Drug Therapy
          Ointments Therapeutic Use
          Wound Care Methods
          Wound Healing Drug Effects
          Ointments Pharmacodynamics
          Neovascularization, Physiologic Drug Effects
          Ulcer Drug Therapy
          Hyperglycemia Complications
          Receptors, Cell Surface Drug Effects
          Stromal Cells Drug Effects
          Animal Studies
          Mice
          Wound Healing Physiology
          Collagen Drug Effects
          Microvascular Density Drug Effects
          Descriptive Statistics
          Comparative Studies
          Treatment Outcomes
          Funding Source
      ab: The mechanism by which moist‐exposed burn ointment (MEBO) promotes wound healing in diabetic foot ulcers (DFUs) remains unclear. To elucidate this mechanism, we investigated the role of stromal cell‐derived factor‐1 (SDF‐1) in MEBO's ability to enhance ulcer healing and neovascularisation using a full‐thickness wound model in hyperglycaemic rats. On post‐wounding days 3, 7, and 14, the wound healing rates were significantly higher in the MEBO group compared to the model group and Vaseline group (p < 0.05). Additionally, the MEBO group exhibited increased epidermal layer thickness and enhanced collagen fibre deposition relative to the model group and Vaseline group (p < 0.05). Furthermore, the number of CD31‐positive cells and microvascular density (MVD) were significantly elevated in the MEBO group compared to the model group and Vaseline group (p < 0.05). Flow cytometric analysis also demonstrated that the proportion of CD45−, CD34+, and VEGFR2+ cells in the wound area of the MEBO‐treated group was significantly higher compared to the model group and Vaseline group. Expression levels of SDF‐1, HIF‐1α were also markedly higher in the MEBO group compared to the model group and Vaseline group (p < 0.05). Finally, a significant increase in double‐positive CXCR4 and CD31 cells was observed exclusively in the MEBO group of hyperglycaemic rats (p < 0.05). These findings suggest that MEBO therapy promotes angiogenesis and accelerates wound healing through activation of the SDF‐1/CXCR4 axis during wound healing in diabetics.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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