Lauric Acid Promotes Esophageal Squamous Cell Carcinoma via GPR84: Lipid Metabolic Dysregulation as a Therapeutic Target.

Various studies have demonstrated metabolic disorders in patients with esophageal squamous cell carcinoma (ESCC), although their potential role in ESCC development remains unclear. Here, we investigated alterations in the serum and fecal metabolomes of 23 ESCC patients and 23 healthy controls using...

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Publicado en:Nutrition & Cancer Vol. 77; no. 9; pp. 1074 - 1089
Autores principales: Shen, Weitao, Li, Nan, Zhang, Anqi, Quan, MengJie, Cao, Jing, Qian, Qiliu, Tang, Derong, Hu, Jian, Lin, Qiang
Formato: pictorial research tables/charts Journal Article
Publicado: Taylor & Francis Ltd 2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2025
      vid: 77
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      pub: Taylor & Francis Ltd
      place: Philadelphia, Pennsylvania
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        10.1080/01635581.2025.2535053
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        atl: Lauric Acid Promotes Esophageal Squamous Cell Carcinoma via GPR84: Lipid Metabolic Dysregulation as a Therapeutic Target.
      aug:
        au:
          Shen, Weitao
          Li, Nan
          Zhang, Anqi
          Quan, MengJie
          Cao, Jing
          Qian, Qiliu
          Tang, Derong
          Hu, Jian
          Lin, Qiang
        affil: Department of Oncology, North China Petroleum Bureau General Hospital, Hebei Medical University, Renqiu, China
      sug:
        subj:
          Fatty Acids Adverse Effects
          Esophageal Squamous Cell Carcinoma Risk Factors
          Receptors, Cell Surface Adverse Effects
          Lipids Metabolism
          Metabolomics
          Risk Assessment
          Tumor Markers, Biological
          Human
          Feces Analysis
          Metabolites
          In Vitro Studies
          Cell Line, Tumor
          Chromatography, High Pressure Liquid
          Signal Transduction
          Linear Regression
          Polymerase Chain Reaction
          Flow Cytometry
          Apoptosis
          Cell Cycle
          Descriptive Statistics
          Mann-Whitney U Test
          Data Analysis Software
          Steroids Metabolism
          Gene Expression
          Cell Viability
          Funding Source
      ab: Various studies have demonstrated metabolic disorders in patients with esophageal squamous cell carcinoma (ESCC), although their potential role in ESCC development remains unclear. Here, we investigated alterations in the serum and fecal metabolomes of 23 ESCC patients and 23 healthy controls using untargeted metabolomics. Further analysis identified lauric acid as a biomarker for ESCC through targeted metabolomics, with validation in 88 ESCC patients and 44 healthy controls. Then, we examined the effects and mechanisms of lauric acid on EC109 cell physiological functions. Significant alterations occurred in serum and fecal samples from ESCC patients versus healthy controls, with lipid metabolites and associated pathways showing the most pronounced changes. Lauric acid was identified as a characteristic altered metabolite and an independent risk factor for ESCC (OR = 1.71, 95% CI: 1.05–2.79, p < 0.05). Mechanistic studies demonstrated that lauric acid promotes EC109 survival, invasion, and migration by interacting with the highly expressed fatty acid receptor GPR84 in ESCC tissues. Our research provides new insights into the relationship between lipid metabolism disorders and ESCC development, suggesting that lauric acid serves as an ESCC risk factor and may offer therapeutic intervention avenues.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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