Anti-colorectal cancer effect and mechanism of a polysaccharide from Ruditapes philippinarum.

Purpose: Colorectal cancer (CRC) is the most common malignant tumor with a persistently high mortality rate worldwide. This study aimed to explore the anti-CRC effect and mechanisms of Ruditapes philippinarum polysaccharides (RPP). Methods: We used HT-29 cells and HT-29 tumor-bearing mice to study t...

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Publicado en:European Journal of Nutrition Vol. 64; no. 6; pp. 1 - 15
Autores principales: Zhang, Xinyu, Liu, Mengyue, Zhang, Xuemei, Du, Zubo, Wei, Yuxi, Li, Fei
Formato: Journal Article
Publicado: Springer Nature Sep2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Sep2025
      vid: 64
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      pub: Springer Nature
      place: New York, New York
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        187803096
        10.1007/s00394-025-03794-8
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        atl: Anti-colorectal cancer effect and mechanism of a polysaccharide from Ruditapes philippinarum.
      aug:
        au:
          Zhang, Xinyu
          Liu, Mengyue
          Zhang, Xuemei
          Du, Zubo
          Wei, Yuxi
          Li, Fei
        affil: https://ror.org/021cj6z65 College of Life Science, Qingdao University, 308 Ningxia Road, 266071, Qingdao, China
      sug:
      ab: Purpose: Colorectal cancer (CRC) is the most common malignant tumor with a persistently high mortality rate worldwide. This study aimed to explore the anti-CRC effect and mechanisms of Ruditapes philippinarum polysaccharides (RPP). Methods: We used HT-29 cells and HT-29 tumor-bearing mice to study the anti-CRC effects and mechanisms of RPP in vitro and in vivo. Results: Animal experiments demonstrated that RPP-treated groups increased expression levels of Bax and cleaved caspase-3 proteins while reducing expression levels of Bcl-2 and Clusterin proteins in mice bearing HT-29 tumors. These results suggested the potential involvement of the mitochondrial apoptotic pathway in HT-29 cells apoptosis. Meanwhile, expression levels of β-catenin and its subsequent target proteins, such as Survivin, C-myc, and Cyclin D1, were suppressed, suggesting that RPP arrested the cell cycle of HT-29 cells in the G0/G1 phase by inhibiting the Wnt/β-catenin pathway. Additionally, RPP suppressed levels of TNF-α, IL-6, and IL-1β, and then alleviated the inflammatory response. Conclusion: Overall, these findings could provide a new insight that RPP are promising polysaccharides to combat CRC as drugs or preventive health products.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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