The possible effect of inflammation on non-suicidal self-injury in adolescents with depression: a mediator of connectivity within corticostriatal reward circuitry.

Non-suicidal self-injury (NSSI) in adolescent depression is a prevalent and clinically significant behavior linked to dysregulated peripheral inflammation and corticostriatal circuitry dysfunction. However, the neuroimmune mechanisms bridging these systems remain poorly understood. Here, we combined...

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Publicado en:European Child & Adolescent Psychiatry Vol. 34; no. 9; pp. 2871 - 2886
Autores principales: Qiao, Dan, Qi, Yirun, Zhang, Xiaoyu, Wen, Yujiao, Huang, Yangxi, Li, Yiran, Liu, Penghong, Li, Gaizhi, Liu, Zhifen
Formato: research tables/charts Journal Article
Publicado: Springer Nature Sep2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Sep2025
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00787-025-02709-6
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        atl: The possible effect of inflammation on non-suicidal self-injury in adolescents with depression: a mediator of connectivity within corticostriatal reward circuitry.
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          Qiao, Dan
          Qi, Yirun
          Zhang, Xiaoyu
          Wen, Yujiao
          Huang, Yangxi
          Li, Yiran
          Liu, Penghong
          Li, Gaizhi
          Liu, Zhifen
        affil: https://ror.org/02vzqaq35 Department of Psychiatry, First Hospital of Shanxi Medical University, No. 85 Jiefang South Road, 030001, Taiyuan, China
      sug:
        subj:
          Self-Injurious Behavior Risk Factors
          Inflammation
          Depression Complications
          Functional Connectivity
          Neuroradiography
          Reward
          Antiinflammatory Agents
          Cytokines
          Human
          Funding Source
          Male
          Female
          Adolescence
          Child
          Young Adult
          Cross Sectional Studies
          China
          Antidepressive Agents
          Psychotherapy
          Nervous System Diseases
          Mental Disorders
          Magnetic Resonance Imaging
          Enzyme-Linked Immunosorbent Assay
          Interleukins
          Analysis of Covariance
          Pearson's Correlation Coefficient
          Fisher's Exact Test
          Descriptive Statistics
          Post Hoc Analysis
          Data Analysis Software
          Confidence Intervals
          Scales
          Adolescent: 13-18 years
          Child: 6-12 years
          Male
          Female
      ab: Non-suicidal self-injury (NSSI) in adolescent depression is a prevalent and clinically significant behavior linked to dysregulated peripheral inflammation and corticostriatal circuitry dysfunction. However, the neuroimmune mechanisms bridging these systems remain poorly understood. Here, we combined peripheral cytokine profiling with static/dynamic functional connectivity (sFC/dFC) analysis to investigate the potential influence of inflammaton on corticostriatal circuit related to NSSI. A set of peripheral blood inflammatory markers and resting-state functional magnetic resonance imaging (rs-fMRI) were collected in depression with NSSI (NSSI+), depression without NSSI (NSSI-), and healthy controls (HC). We first ascertain group differences in level of pro- and anti-inflammatory cytokines. And using ventral/dorsal striatal seeds, we compared whole-brain, voxel-wise sFC and dFC differences across three groups. Further, we tested the mediation effects of connectivity in the association between inflammatory markers and NSSI frequency. NSSI+ group exhibited elevated pro-inflammatory cytokines (C-reactive protein (CRP), interleukin (IL)-1, and IL-6) whereas reduced anti-inflammatory cytokines (IL-10), compared to NSSI− and HC. Neuroimaging analysis revealed corticostriatal dysconnectivity mainly characterized by static hyperconnectivity between dorsal striatum and thalamus, dynamic instability in dorsal striatum-lingual pathways, and dynamic rigidity in ventral striatum-prefrontal/temporal/occipital gyrus circuits. Critically, sFC of dorsal striatum-thalamus and dFC of dorsal striatum-lingual gyrus mediated the prospective association between altered CRP and NSSI frequency, establishing corticostriatal circuits as conduits for inflammatory effects on NSSI. By bridging molecular psychiatry with circuit neuroscience, this work advances precision management of NSSI in adolescent depression, prioritizing biomarker-driven strategies to disrupt neuroimmune maladaptation.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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