Marine ω-3 PUFA Supplementation Enhances FFAR4 Activation and Reduces Inflammatory Markers in PBMC of Subjects with Obesity: A Randomized Controlled Trial (EPICO).

Background: It is widely accepted that low-grade chronic inflammation in obesity worsens the metabolic state and threatens patients' lives in a long-term manner. In fact, diet therapy is the first-line treatment in which relevant nutrients such as the omega-3 polyunsaturated fatty acids (ω-3 PUFA) m...

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Publicado en:Nutrients Vol. 17; no. 23; pp. 3630 - 3659
Autores principales: Reyes-Pérez, Samantha Desireé, Cambron-Mora, Diego, Torres-Vanegas, Joel, Mojica-Zamudio, Karla Lizette, Olaez-Ramos, Carolina Elizabeth, Lauriano-Rivera, Ramon Gerardo, Rivera-Valdés, Juan J., Torres-Castillo, Nathaly, Valencia-Sosa, Evelyn, Rodriguez-Echevarria, Roberto, Martinez-Lopez, Erika
Formato: research tables/charts randomized controlled trial Journal Article
Publicado: MDPI Dec2025
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2025
      vid: 17
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      pub: MDPI
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        10.3390/nu17233630
        190517625
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        atl: Marine ω-3 PUFA Supplementation Enhances FFAR4 Activation and Reduces Inflammatory Markers in PBMC of Subjects with Obesity: A Randomized Controlled Trial (EPICO).
      aug:
        au:
          Reyes-Pérez, Samantha Desireé
          Cambron-Mora, Diego
          Torres-Vanegas, Joel
          Mojica-Zamudio, Karla Lizette
          Olaez-Ramos, Carolina Elizabeth
          Lauriano-Rivera, Ramon Gerardo
          Rivera-Valdés, Juan J.
          Torres-Castillo, Nathaly
          Valencia-Sosa, Evelyn
          Rodriguez-Echevarria, Roberto
          Martinez-Lopez, Erika
        affil: Instituto de Nutrigenética y Nutrigenómica Traslacional, Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara 44340, Jalisco, Mexico
      sug:
        subj:
          Obesity Drug Therapy
          Fatty Acids, omega-3 Therapeutic Use
          Inflammation Blood
          Biological Markers Blood
          Receptors, G-Protein-Coupled Metabolism
          Dietary Supplementation
          Treatment Outcomes
          Human
          Funding Source
          Male
          Female
          Double-Blind Studies
          Randomized Controlled Trials
          Random Assignment
          Adult
          Middle Age
          Aged
          Body Mass Index
          Waist Circumference
          Nutritional Assessment
          Physical Activity
          Blood Specimen Collection
          Proteins
          Blotting, Western
          RNA
          Cytokines
          Descriptive Statistics
          Data Analysis Software
          Paired T-Tests
          Analysis of Variance
          Post Hoc Analysis
          Friedman Test
          Spearman's Rank Correlation Coefficient
          Linear Regression
          Chi Square Test
          Questionnaires
          Triglycerides
          Lipoproteins, LDL Cholesterol
          Insulin Sensitivity Prevention and Control
          Lipoproteins, HDL Cholesterol
          Correlational Studies
          Gene Expression
          Transferases
          Tumor Necrosis Factor
          Adult: 19-44 years
          Middle Aged: 45-64 years
          Aged: 65+ years
          Male
          Female
      ab: Background: It is widely accepted that low-grade chronic inflammation in obesity worsens the metabolic state and threatens patients' lives in a long-term manner. In fact, diet therapy is the first-line treatment in which relevant nutrients such as the omega-3 polyunsaturated fatty acids (ω-3 PUFA) must be adequately consumed to counteract the established inflammation. In particular, eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) have been identified as agonists of cellular receptors, including the free fatty acid receptor 4 (FFAR4), which regulates anti-inflammatory pathways associated with enhanced insulin sensitivity. However, the expression and activation of this receptor in peripheral blood mononuclear cells (PBMC) remains poorly investigated in humans. Objective: This study aimed to evaluate the effect of a diet supplemented with marine ω-3 PUFA on FFAR4 receptor activation and inflammatory markers in peripheral blood mononuclear cells in subjects with obesity. Methodology: A double-blind, randomized clinical trial (NCT05068557) was conducted over two months (eight weeks) in 55 obese individuals (aged 25–59 years). Participants were randomly assigned to one of two groups: an active placebo group (1.6 g/day of alpha-linolenic acid) or a marine ω-3 group (1080 mg of EPA and 720 mg of DHA). Both groups followed a dietary regimen with progressive calorie restriction (−200 kcal/day during weeks 0–4 and −400 kcal/day during weeks 4–8) in addition to supplementation. Results: Following the intervention, both groups showed significant improvements in body composition and biochemical parameters. Supplementation with EPA and DHA enhanced FFAR4 activation at the end of the intervention. Moreover, there was a reduction in the expression of JNK and IKKβ genes, as well as in serum levels of TNF-α, IL-6, and IL-18. In contrast, IL-10 levels increased significantly both within and between the groups. Conclusions: Marine ω-3 PUFA supplementation, in the context of a dietary intervention, promotes FFAR4 activation, thereby contributing to the modulation of the inflammatory response in human PBMC.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        randomized controlled trial
        Journal Article
      ougenre: Article
    language: English
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