Effect of a Nutraceutical Combination on Oxidative Stress Biomarkers in Healthy Subjects and Patients with Alzheimer's Disease.

Background/Objectives: Advanced glycation end products (AGEs) and oxidative stress increase with aging and are implicated in Alzheimer's disease (AD). We developed an anti-glycation blend using LC-MS-based screening and assessed its effects on oxidative and glycation-related biomarkers in humans. Me...

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Publicado en:Nutrients Vol. 18; no. 5; pp. 789 - 802
Autores principales: Jastrząb, Rafał, Małecki, Andrzej, Kmiecik-Małecka, Elżbieta, Gorzkowska, Agnieszka, Kubas, Kamil, Widłak-Kargul, Justyna, Wolman, Damian, Matkiewicz, Katarzyna, Nowacka-Chmielewska, Marta, Liśkiewicz, Daniela, Grabowska, Konstancja, Grabowski, Mateusz, Pondel, Natalia, Początek, Gabriela, Kłodowska, Gabriela, Mytych, Jennifer
Formato: research tables/charts randomized controlled trial Journal Article
Publicado: MDPI Mar2026
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Mar2026
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      pub: MDPI
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        atl: Effect of a Nutraceutical Combination on Oxidative Stress Biomarkers in Healthy Subjects and Patients with Alzheimer's Disease.
      aug:
        au:
          Jastrząb, Rafał
          Małecki, Andrzej
          Kmiecik-Małecka, Elżbieta
          Gorzkowska, Agnieszka
          Kubas, Kamil
          Widłak-Kargul, Justyna
          Wolman, Damian
          Matkiewicz, Katarzyna
          Nowacka-Chmielewska, Marta
          Liśkiewicz, Daniela
          Grabowska, Konstancja
          Grabowski, Mateusz
          Pondel, Natalia
          Początek, Gabriela
          Kłodowska, Gabriela
          Mytych, Jennifer
        affil: Research and Development Center, Olimp Laboratories Sp. z o.o. 39-200 Dębica, Poland
      sug:
        subj:
          Alzheimer's Disease Diet Therapy
          Functional Food Therapeutic Use
          Oxidative Stress Evaluation
          Biological Markers
          Treatment Outcomes
          Dietary Supplements
          Glycation End Products, Advanced
          Aging
          Funding Source
          Human
          Male
          Female
          Aged
          Aged, 80 and Over
          Descriptive Statistics
          Randomized Controlled Trials
          Random Assignment
          Liquid Chromatography-Mass Spectrometry
          Double-Blind Studies
          Analysis of Variance
          Post Hoc Analysis
          Prospective Studies
          Enzyme-Linked Immunosorbent Assay
          Data Analysis Software
          Colorimetry
          Aged: 65+ years
          Aged, 80 & over
          Male
          Female
      ab: Background/Objectives: Advanced glycation end products (AGEs) and oxidative stress increase with aging and are implicated in Alzheimer's disease (AD). We developed an anti-glycation blend using LC-MS-based screening and assessed its effects on oxidative and glycation-related biomarkers in humans. Methods: Twelve candidate compounds were screened in a BSA–glucose model using LC-MS peptide mapping to quantify lysine glycation and rank inhibitory activity. The top candidates were combined into a three-compound blend (quercetin, rutin, genistein). In a randomized, double-blind, placebo-controlled 3-month trial, older healthy adults (n = 30) and individuals with AD (n = 30) received anti-AGE blend (n = 15 in older group and n = 15 in AD group) or placebo (n = 15 in older group and n = 15 in AD group). Serum malondialdehyde and urinary Nε-(carboxymethyl)lysine were measured pre–post intervention. Pre/post and between-arm comparisons within each population were performed using REML ANOVA with Tukey post hoc tests. Serum MDA (malondialdehyde) and urinary CML (Nε-(carboxymethyl)lysine) were prespecified biomarker outcomes and are reported here as co-primary biomarker endpoints. No formal a priori sample size calculation was performed; the study size was feasibility-based. Results: LC-MS screening identified genistein, quercetin, and rutin as the most consistent inhibitors of glucose-driven BSA glycation. In older healthy adults, serum MDA decreased after anti-AGE supplementation (p < 0.001) and differed from the placebo (p < 0.01), while no change was observed within the placebo group (ns). In the AD cohort, MDA did not change significantly from baseline within either arm (ns), but post-intervention MDA was lower in anti-AGE than in the placebo (p < 0.05). Urinary CML was unchanged in older healthy adults (ns in both arms), whereas in AD, it decreased after anti-AGE supplementation (p < 0.01) and differed from the placebo (p < 0.05). Conclusions: A screening-guided anti-glycation blend supplementation was associated with changes in selected biomarkers in humans: MDA decreased across cohorts, while CML decreased selectively in AD. Larger trials with extended biomarker panels and LC–MS/MS confirmation are warranted.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        randomized controlled trial
        Journal Article
      ougenre: Article
    language: English
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