Venetoclax Drives Significant Shifts in BIM and BCL-2 but not MCL-1 Gene Expression in a Mouse Model of Acute Lymphoblastic Leukemia.

Background: T and B acute lymphoblastic leukemia (T, B-ALL) has seen improved survival rates with intensified chemotherapy, but therapy-resistant or refractory ALL remains a significant clinical challenge. This study examined the inhibitory effects of Venetoclax drug on the apoptosis gene to explore...

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Publicado en:Middle East Journal of Cancer Vol. 17; no. 1; pp. 9 - 20
Autores principales: Tari, Kaveh, Abroun, Saied
Formato: pictorial research tables/charts Journal Article
Publicado: Middle East Journal of Cancer Jan2026
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jan2026
      vid: 17
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      pub: Middle East Journal of Cancer
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        193694179
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        10.30476/mejc.2025.104911.2214
        193694179
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        atl: Venetoclax Drives Significant Shifts in BIM and BCL-2 but not MCL-1 Gene Expression in a Mouse Model of Acute Lymphoblastic Leukemia.
      aug:
        au:
          Tari, Kaveh
          Abroun, Saied
        affil: Department of Hematology, Faculty of Medical Sciences, Tarbiat Modares University, Tehran, Iran
      sug:
        subj:
          Leukemia, Lymphocytic, Acute Drug Therapy
          Antineoplastic Agents Administration and Dosage
          Gene Expression Profiling
          Proteins
          Models, Biological
          Apoptosis
          Drug Efficacy
          Human
          Animal Studies
          Mice
          Descriptive Statistics
          Experimental Studies
          Reverse Transcriptase Polymerase Chain Reaction
          Prospective Studies
          RNA
          Spectrophotometry
          Flow Cytometry
          Data Analysis Software
          T-Tests
      ab: Background: T and B acute lymphoblastic leukemia (T, B-ALL) has seen improved survival rates with intensified chemotherapy, but therapy-resistant or refractory ALL remains a significant clinical challenge. This study examined the inhibitory effects of Venetoclax drug on the apoptosis gene to explore its potential as a novel therapeutic approach for treating human T, B-ALL. Method: This was a preclinical experimental study. Firstly, samples were collected from a leukemia patient, followed by isolating blast cells that were injected into primary mice. The sample was obtained from mice in 1, 8 and 14 days; the CD45 human was quantified using flow cytometry to confirm the development of leukemia. Spleen cells from the primary mice were isolated and injected into the secondary mice. After 14 days, the Venetoclax drug was administered to the mouse models for 21 days. Subsequently, mouse spleen cells were gathered, and the expression of genes associated with apoptosis was assessed. A two-tailed t-test was performed to compare the expression of apoptotic genes between the control and Venetoclax-treated groups. Results: Our findings indicated a decrease in the expression of the B-cell lymphoma 2 (BCL-2) gene, while the expression of the BCL-2-interacting mediator (BIM) of cell death gene exhibited an augmentation. The level of expression of the MCL-1 (Myeloid leukemia 1) gene did not display any significant divergence compared with the control group. Conclusion: Venetoclax drug shows potential therapeutic potential in B and T-ALL, increasing BIM expression and decreasing BCL-2, but further investigation and clinical trial studies are needed.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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