Advance in the mechanism and treatment of autonomic dysfunction in α-synucleinopathies.

α-Synucleinopathies are a group of neurodegenerative diseases characterized by the abnormal aggregation of α-synuclein (α-Syn), including Parkinson's disease (PD), multiple system atrophy (MSA), dementia with Lewy bodies (DLB), and pure autonomic failure (PAF). Autonomic dysfunction is one of the mo...

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Publicado en:Chinese Journal of Contemporary Neurology & Neurosurgery Vol. 26; no. 4; pp. 352 - 361
Autores principales: TANG, Liu-feng, ZHAO, Zhen-bo, GUAN, Ai, ZHANG, Xiao-yu, LIU, Xiao-yun, WANG, Hua-long
Formato: review Journal Article
Publicado: Chinese Journal of Contemporary Neurology & Neurosurgery Apr2026
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2026
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      pub: Chinese Journal of Contemporary Neurology & Neurosurgery
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        10.3969/j.issn.1672-6731.2026.04.003
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        atl: Advance in the mechanism and treatment of autonomic dysfunction in α-synucleinopathies.
      aug:
        au:
          TANG, Liu-feng
          ZHAO, Zhen-bo
          GUAN, Ai
          ZHANG, Xiao-yu
          LIU, Xiao-yun
          WANG, Hua-long
        affil: Department of Neurology, Renji Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200127, China
      sug:
        subj:
          Synucleinopathies Complications
          Autonomic Nervous System Diseases Physiopathology
          Autonomic Nervous System Diseases Therapy
          Synucleins Metabolism
          Nerve Tissue Proteins Metabolism
          Parkinson Disease Complications
          Multiple System Atrophy Complications
          Lewy Body Disease Complications
          Autonomic Nervous System Diseases Drug Therapy
          Polymers
          Midodrine Therapeutic Use
          Norepinephrine Therapeutic Use
          Serotonin Uptake Inhibitors Therapeutic Use
          Transcranial Magnetic Stimulation
          Vagal Stimulation
          Autonomic Nervous System Diseases Rehabilitation
          Acupuncture
          Immunotherapy
          Antibodies, Monoclonal Therapeutic Use
      ab: α-Synucleinopathies are a group of neurodegenerative diseases characterized by the abnormal aggregation of α-synuclein (α-Syn), including Parkinson's disease (PD), multiple system atrophy (MSA), dementia with Lewy bodies (DLB), and pure autonomic failure (PAF). Autonomic dysfunction is one of the most common non-motor symptoms in α-synucleinopathies, yet its underlying mechanisms remain unclear. α-Synucleinopathies exhibit the significant heterogeneity, and progress in developing novel therapeutic strategies remains limited. This review systemly explores the pathogenesis of autonomic dysfunction in α-synucleinopathies, with a focus on α-Syn conformational abnormalities, the peripheralcentral prion-like spread model, and the mechanisms by which α-Syn aggregates damage peripheral autonomic nerves. Furthermore, it summarizes recent advancements in drug therapies, non-pharmacological therapies, and α-Syn-targeted therapies, aiming to provide new insights for clinical practice and future research on α-synucleinopathies.
      pubtype: Academic Journal
      doctype:
        review
        Journal Article
      ougenre: Article
    language: Chinese
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