Juglone Attenuates CCl₄‐Induced Hepatic Injury in Mice With Associated Modulation of Oxidative Stress and Inflammasome‐Related mRNA Expression.

Carbon tetrachloride–induced liver injury is a well‐established model of toxic hepatitis, characterized by marked oxidative and inflammatory damage. It remains a relevant experimental system because broadly effective therapeutic options are still limited. Juglone, a natural naphthoquinone derived fr...

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Published in:BioMed Research International Vol. 2026; pp. 1 - 13
Main Authors: Sohail, Aamir, Sufyan, Raza, Asim, Muhammad, Khan, Mehroz, Altaf, Aleena, Haq, Muhammad Ehsan Ul, Arshad, Maimoona, Khalil, Atif Ali Khan, Ullah, Imran, Banerjee, Baisakhi
Format: pictorial research tables/charts Journal Article
Published: Wiley-Blackwell 7/21/2026
Online Access:View this record in EBSCOhost
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      dt: 7/21/2026
      vid: 2026
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        195519890
        195519890
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        10.1155/bmri/8905392
        195519890
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        atl: Juglone Attenuates CCl₄‐Induced Hepatic Injury in Mice With Associated Modulation of Oxidative Stress and Inflammasome‐Related mRNA Expression.
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          Sohail, Aamir
          Sufyan, Raza
          Asim, Muhammad
          Khan, Mehroz
          Altaf, Aleena
          Haq, Muhammad Ehsan Ul
          Arshad, Maimoona
          Khalil, Atif Ali Khan
          Ullah, Imran
          Banerjee, Baisakhi
        affil: Department of Biochemistry,, Faculty of Biological Sciences,, Quaid-i-Azam University,, Islamabad, Pakistan, qau.edu.pk
      sug:
        subj:
          Hydrocarbons, Chlorinated
          Liver Diseases Chemically Induced
          Liver Diseases Drug Therapy
          Oxidative Stress Drug Effects
          Inflammation Mediators Drug Effects
          RNA, Messenger Drug Effects
          Gene Expression
          Inflammation Prevention and Control
          Quinones Analysis
          Plants, Medicinal
          Plant Extracts Analysis
          Animal Studies
          Mice
          Biological Products
          Models, Biological
          Experimental Studies
          Staining and Labeling
          Reverse Transcriptase Polymerase Chain Reaction
          Liver Injuries
          Apoptosis
          Endoplasmic Reticulum Stress
          Autophagy
          Weight Loss
          Hyperlipidemia
          Transcription Factors
          Funding Source
          Laboratories
          Pakistan
          Biochemistry
          Biological Assay
          Molecular Biology
          Data Analysis Software
          One-Way Analysis of Variance
          Post Hoc Analysis
      ab: Carbon tetrachloride–induced liver injury is a well‐established model of toxic hepatitis, characterized by marked oxidative and inflammatory damage. It remains a relevant experimental system because broadly effective therapeutic options are still limited. Juglone, a natural naphthoquinone derived from Reynoutria japonica, has demonstrated antioxidant and anti‐inflammatory potential. However, mouse evidence combining standard liver injury measures with stress‐ and cell‐death gene profiling remains limited. Building on juglone studies in injury and fibrosis, we tested juglone in a CCl4 model. Male C57BL/6 mice were assigned to normal, vehicle, CCl₄, silymarin (200 mg/kg), and juglone (8 mg/kg) groups, and CCl₄ (in corn oil) was administered intraperitoneally. Endpoints included body weight, serum liver enzymes and lipid profile, H&E histology, and RT‐qPCR panels for inflammatory, pyroptosis‐associated, ER stress and autophagy‐linked, apoptotic, oxidative‐stress, and profibrotic transcripts. CCl₄ exposure was associated with weight loss, enzyme elevation, dyslipidemia, and architectural disruption, whereas juglone treatment was associated with improved biochemical indices and histology alongside coordinated downregulation of inflammatory/pyroptosis‐related transcripts (including Il-1β, Il-6, Nlrp3, and Gsdmd) and moderation of ER stress/autophagy and apoptosis‐linked transcripts, with downregulation of profibrotic transcripts (Acta2, Mmp2, and Tgfβ1). In this mouse CCl₄ model, juglone treatment was associated with reduced serum and tissue injury signatures together with changes in stress and inflammation‐linked transcriptional programs. Because the molecular findings are primarily based on RT‐qPCR, these results are best interpreted as gene‐level associations compatible with modulation of oxidative stress, inflammasome‐ and pyroptosis‐related mRNA expression, ER stress responses, apoptosis‐linked transcripts, and remodeling‐associated transcripts, rather than as confirmed pathway inhibition or established antifibrotic activity. These data support juglone as a hepatoprotective candidate and identify protein‐level and functional validation as important next steps.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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