Recovery of associative function following early amygdala lesions in rats.

Adult rats with amygdala lesions made at either Postnatal Day (PND) 10 or PND40 were tested on a series of reversal tasks that tap the ability to form stimulus–reward associations. PND40 rats were significantly impaired relative to both controls and PND10 rats on learning rate of the original discr...

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Publicado en:Behavioral Neuroscience Vol. 115; no. 1; pp. 154 - 165
Autores principales: Higley, M. J., Hermer-Vazquez, L., Levitsky, D. A.
Formato: Artículo
Publicado: American Psychological Association February 2001
Materias:
Acceso en línea:Ver este registro en EBSCOhost
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      pub: American Psychological Association
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        atl: Recovery of associative function following early amygdala lesions in rats.
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          Higley, M. J.
          Hermer-Vazquez, L.
          Levitsky, D. A.
      su:
        Laboratory rats
        Amygdaloid body
        Discrimination learning
        Brain function localization
      sug:
        subj:
          Laboratory rats
          Amygdaloid body
          Discrimination learning
          Brain function localization
      ab: Adult rats with amygdala lesions made at either Postnatal Day (PND) 10 or PND40 were tested on a series of reversal tasks that tap the ability to form stimulus–reward associations. PND40 rats were significantly impaired relative to both controls and PND10 rats on learning rate of the original discrimination and subsequent reversals. Analyses of discrete learning phases revealed that the impairment was specific to the postchance phase. The PND10 group was not impaired relative to controls on any measure. These results confirm prior findings that amygdala lesions sustained in adulthood impair the formation of stimulus–reward associations. They also demonstrate that substantial sparing or recovery of function is possible when the lesion is made during early development. Furthermore, the findings support the view that behavioral recovery may be more likely if the lesion is sustained near the time of peak synaptogenesis. Reprinted by permission of the publisher.
      pubtype: Academic Journal
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    language: English
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