Fucoidan Inhibited 4T1 Mouse Breast Cancer Cell Growth In Vivo and In Vitro Via Downregulation of Wnt/β-Catenin Signaling.

Fucoidan is a sulfated polysaccharide derived from brown algae and is known to possess anticancer properties. However, the relationship between fucoidan and β-catenin, one of the key components of the Wnt signaling pathway, in mouse breast cancer remains poorly characterized. In this study, mouse br...

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Publicado en:Nutrition & Cancer Vol. 65; no. 3; pp. 460 - 469
Autores principales: Xue, Meilan, Ge, Yinlin, Zhang, Jinyu, Liu, Yongchao, Wang, Qing, Hou, Lin, Zheng, Zheng
Formato: research Journal Article
Publicado: Taylor & Francis Ltd Apr2013
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2013
      vid: 65
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      pub: Taylor & Francis Ltd
      place: Philadelphia, Pennsylvania
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        86409267
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        10.1080/01635581.2013.757628
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        atl: Fucoidan Inhibited 4T1 Mouse Breast Cancer Cell Growth In Vivo and In Vitro Via Downregulation of Wnt/β-Catenin Signaling.
      aug:
        au:
          Xue, Meilan
          Ge, Yinlin
          Zhang, Jinyu
          Liu, Yongchao
          Wang, Qing
          Hou, Lin
          Zheng, Zheng
        affil: Department of Biochemistry and Molecular Biology, Medical College, Qingdao University, Qingdao, Shandong Province, China
      sug:
        subj:
          Cell Physiology
          Nutrition
          Animal Studies
          Mice
          In Vivo Studies
          In Vitro Studies
          Cell Cycle
          Blotting, Western
          Biological Assay
          Immunohistochemistry
          Data Analysis
          Descriptive Statistics
          T-Tests
      ab: Fucoidan is a sulfated polysaccharide derived from brown algae and is known to possess anticancer properties. However, the relationship between fucoidan and β-catenin, one of the key components of the Wnt signaling pathway, in mouse breast cancer remains poorly characterized. In this study, mouse breast cancer cells (4T1) were exposed to fucoidan to investigate the relationship between fucoidan and the Wnt/β-catenin signaling pathway in vivo and in vitro. We found that fucoidan significantly inhibited cell growth, increased cell death, and induced G1 cell cycle arrest in 4T1 cells. Fucoidan also reduced β-catenin expression and T cell factor/lymphoid-enhancing factor reporter activity. Furthermore, fucoidan downregulated the expression of downstream target genes such as c-myc, cyclin D1, and survivin. Intraperitoneal injection of fucoidan in tumor-bearing mice reduced the tumor volume and weight. Fucoidan induced aberrant downregulation of β-catenin in tumor tissues with a significant increase in apoptosis. Thus, our data suggested that fucoidan exerts its anticancer activity through downregulation of Wnt/β-catenin signaling. Fucoidan may be an effective therapy for the chemoprevention and treatment of mouse breast cancer.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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