High dose ascorbic acid does not reverse central sympathetic overactivity in chronic heart failure.

Summary What is known and Objective: The increased central sympathetic activity typically associated with chronic heart failure (CHF) is probably mediated by formation of reactive oxygen species (ROS) in the brain. Our objective was to undertake a trial to test our hypothesis that administration of...

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Published in:Journal of Clinical Pharmacy & Therapeutics Vol. 36; no. 5; pp. 546 - 553
Main Authors: Gomes, M. E., El Messaoudi, S., Lenders, J. W. M., Bellersen, L., Verheugt, F. W. A., Smits, P., Tack, C. J.
Format: research tables/charts randomized controlled trial Journal Article
Published: Wiley-Blackwell Oct2011
Online Access:View this record in EBSCOhost
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      dt: Oct2011
      vid: 36
      iid: 5
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        104685566
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        10.1111/j.1365-2710.2010.01205.x
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        atl: High dose ascorbic acid does not reverse central sympathetic overactivity in chronic heart failure.
      aug:
        au:
          Gomes, M. E.
          El Messaoudi, S.
          Lenders, J. W. M.
          Bellersen, L.
          Verheugt, F. W. A.
          Smits, P.
          Tack, C. J.
        affil: Department of Cardiology
      sug:
        subj:
          Ascorbic Acid Administration and Dosage
          Heart Failure Drug Therapy
          Sympathetic Nervous System Drug Effects
          Prospective Studies
          Randomized Controlled Trials
          Double-Blind Studies
          Crossover Design
          Ascorbic Acid Blood
          Heart Rate Variability
          Norepinephrine Blood
          Human
          Catecholamines Blood
          Electrodiagnosis
          Data Analysis Software
          Chromatography, High Pressure Liquid
          Power Analysis
          T-Tests
          Parametric Statistics
          Matched-Pair Analysis
          Nonparametric Statistics
          Wilcoxon Rank Sum Test
          Pearson's Correlation Coefficient
          Male
          Female
          Middle Age
          Treatment Outcomes
          Reactive Oxygen Species Drug Effects
          Middle Aged: 45-64 years
          Male
          Female
      ab: Summary What is known and Objective: The increased central sympathetic activity typically associated with chronic heart failure (CHF) is probably mediated by formation of reactive oxygen species (ROS) in the brain. Our objective was to undertake a trial to test our hypothesis that administration of the well-known antioxidant and ROS scavenger ascorbic acid, would reverse or reduce the sympathetic overactivity in CHF patients. Methods: In a prospective, randomized, placebo-controlled, double-blind, cross-over trial, 11 CHF patients were treated with ascorbic acid 2 g/day or placebo for 3 days. At the end of each treatment period, sympathetic nervous system activity was measured by microneurography for direct muscle sympathetic nerve activity (MSNA) recording, analysis of heart rate variability (HRV) and measurement of plasma norepinephrine concentrations. Results: During ascorbic acid administration, plasma vitamin C levels were higher than during placebo (74·9 ± 6·0 μmol/L vs. 54·8 ± 4·6 μmol/L, P = 0·03). Ascorbic acid had no effect on sympathetic activity: MSNA (ascorbic acid: 66·8 ± 3·3 vs. placebo 66·9 ± 3·2 bursts/100 beats, P = 0·98). In addition, HRV and plasma norepinephrine levels did not differ. What is new and Conclusion: Short-term administration of the antioxidant ascorbic acid in CHF patients does not reverse the increased sympathetic activity as measured by microneurography, HRV and plasma norepinephrine levels. The use of higher oral dosages seems not feasible due to accompanying side effects.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        randomized controlled trial
        Journal Article
      ougenre: Article
    language: English
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