Could CD4 capture by CD8+ T cells play a role in HIV spreading?

CD8(+) T cells have been shown to capture plasma membrane fragments from target cells expressing their cognate antigen, a process termed 'trogocytosis'. Here, we report that human CD4, the Human Immunodeficiency Virus (HIV) receptor, can be found among the proteins transferred by trogocytosis. CD4 i...

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Publicado en:Journal of Biomedicine & Biotechnology pp. 907371 - 907372
Autores principales: Aucher A, Puigdomènech I, Joly E, Clotet B, Hudrisier D, Blanco J
Formato: research Journal Article
Publicado: Wiley-Blackwell 2010
Acceso en línea:Ver este registro en EBSCOhost
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        atl: Could CD4 capture by CD8+ T cells play a role in HIV spreading?
      aug:
        au:
          Aucher A
          Puigdomènech I
          Joly E
          Clotet B
          Hudrisier D
          Blanco J
        affil: Institut de Pharmacologie et de Biologie Structurale (CNRS, IPBS), 205 route de Narbonne, 31077 Toulouse, France.
      sug:
        subj:
          Antigens, CD4 Immunology
          HIV Infections
          HIV Infections Immunology
          Human Immunodeficiency Virus Physiology
          T Lymphocytes Immunology
          Animal Studies
          Cells
          Flow Cytometry
          HLA Antigens Immunology
          Human
          Human Immunodeficiency Virus Immunology
          Immune System
          Mice
          T Lymphocytes
          Viruses
          Viruses Metabolism
      ab: CD8(+) T cells have been shown to capture plasma membrane fragments from target cells expressing their cognate antigen, a process termed 'trogocytosis'. Here, we report that human CD4, the Human Immunodeficiency Virus (HIV) receptor, can be found among the proteins transferred by trogocytosis. CD4 is expressed in a correct orientation after its capture by CD8(+) T cells as shown by its detection using conformational antibodies and its ability to allow HIV binding on recipient CD8(+) T cells. Although we could not find direct evidence for infection of CD8(+) T cells having captured CD4 by HIV, CD4 was virologically functional on these cells as it conferred on them the ability to undergo syncytia formation induced by HIV-infected MOLT-4 cells. Our results show that acquisition of CD4 by CD8(+) T cells via trogocytosis could play a previously unappreciated role for CD8(+) T cells in HIV spreading possibly without leading to their infection.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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