Sciatic inflammatory neuritis: a non-surgical technique.

Many animal models of neuropathic pain involve both surgery and neural trauma. Chacur et al described the surgical placement of perisciatic gelfoam and a cannula through which zymosan was introduced after healing. We report a simpler percutaneous technique by which to deliver zymosan to the sciatic...

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Publicado en:Journal of Neuropathic Pain & Symptom Palliation Vol. 1; no. 4; pp. 3 - 12
Autores principales: Zitron IM, Hartrick CT
Formato: pictorial research tables/charts Journal Article
Publicado: Taylor & Francis Ltd 2005
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2005
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      pub: Taylor & Francis Ltd
      place: Philadelphia, Pennsylvania
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        106202659
        106202659
        2009303836
        10.3109/j426v01n04_02
        106202659
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        atl: Sciatic inflammatory neuritis: a non-surgical technique.
      aug:
        au:
          Zitron IM
          Hartrick CT
        affil: John D. Dingell VA Center, 4646 John Road, Detroit, MI 48201; izitron@med.wayne.edu
      sug:
        subj:
          Glucans Administration and Dosage
          Neuritis Drug Therapy
          Sciatic Nerve Physiopathology
          Allodynia
          Analysis of Variance
          Animal Studies
          Control Group
          Data Analysis Software
          Experimental Studies
          Funding Source
          Mann-Whitney U Test
          Microscopy
          Models, Biological
          Rats
          Repeated Measures
          Sciatic Nerve Pathology
          Spinal Cord Pathology
      ab: Many animal models of neuropathic pain involve both surgery and neural trauma. Chacur et al described the surgical placement of perisciatic gelfoam and a cannula through which zymosan was introduced after healing. We report a simpler percutaneous technique by which to deliver zymosan to the sciatic nerve and in the rat. Using a nerve stimulator we located the sciatic nerve and delivered either zymosan in incomplete Freunds (IFA) or IFA alone. Animals were tested for tactile allodynia at baseline and for up to 12 days post-treatment. Mean baseline withdrawal values did not differ between groups. Zymosan-treated rats showed decreased paw withdrawal thresholds (PWT) from day 3 [day 0: 14.3 +/- 0.52 g; day 3: 8.5 +/- 0.42 g; p < 0.05]. At day 12 there was bilateral tactile allodynia in animals that received zymosan [ipsilateral PWT: 4.9 +/- 1.01 g; contralateral: 7.2 +/- 1.35 g]. Statistically significant changes were observed only for zymosan-treated rats. We extended the observations to approximately 70 days, at which time allodynia was still robust. Both astrocyte activation and IL-1â expression in the ipsilateral spinal cord of persistently allodynic rats appeared to be greater than that in their counterparts that had recovered. This approach represents a simple, non-surgical procedure by which to induce chronic inflammatory neuritis. It thus lends itself to the study of both chronic pain and the prodromal phase.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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