Latent herpes simplex virus in human trigeminal ganglia. Detection of an immediate early gene "anti-sense" transcript by in situ hybridization.

We used in situ hybridization to study the expression of herpes simplex virus type 1 genes during latent infections of human sensory ganglia. Trigeminal ganglia were recovered at autopsy from 24 subjects with no evidence of an active herpetic infection. These ganglia were hybridized to 35S-labeled s...

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Publicado en:New England Journal of Medicine Vol. 317; no. 23; pp. 1427 - 1433
Autores principales: Croen, K D, Ostrove, J M, Dragovic, L J, Smialek, J E, Straus, S E
Formato: research Journal Article
Publicado: New England Journal of Medicine 12/3/87
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 12/3/87
      vid: 317
      iid: 23
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      pub: New England Journal of Medicine
      place: Waltham, Massachusetts
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        atl: Latent herpes simplex virus in human trigeminal ganglia. Detection of an immediate early gene "anti-sense" transcript by in situ hybridization.
      aug:
        au:
          Croen, K D
          Ostrove, J M
          Dragovic, L J
          Smialek, J E
          Straus, S E
        affil: Medical Virology Section, National Institute of Allergy and Infectious Diseases, Bethesda, Md
      sug:
        subj:
          Proteins
          Genes
          RNA
          Herpes Simplex Microbiology
          Herpesviruses
          Trigeminal Nerve Microbiology
          Ganglia, Sensory Microbiology
          Middle Age
          Herpesviruses Immunology
          Adult
          Enzymes
          Aged
          Child
          Nucleic Acid Hybridization
          Adolescence
          RNA Analysis
          Antibodies, Viral Analysis
          Human
          Middle Aged: 45-64 years
          Adult: 19-44 years
          Aged: 65+ years
          Child: 6-12 years
          Adolescent: 13-18 years
      ab: We used in situ hybridization to study the expression of herpes simplex virus type 1 genes during latent infections of human sensory ganglia. Trigeminal ganglia were recovered at autopsy from 24 subjects with no evidence of an active herpetic infection. These ganglia were hybridized to 35S-labeled single-stranded RNA probes spanning 72 percent of the herpes simplex genome. In the ganglia of 16 subjects, 0.2 to 4.3 percent of the neuronal cells contained abundant nuclear signals for viral RNA. Ganglia from three patients with low or undetectable levels of antibodies to herpes simplex type 1 lacked viral RNA signals, whereas the ganglia from all of six patients with elevated antibody titers showed viral RNA signals. Transcription was detected only from the region of the viral genome containing a gene that encodes an immediate early protein known as the infected-cell protein number zero (ICP0). However, normal ("sense") transcripts of this gene, which are prominent in an acute infection, were not detected. In contrast, a novel transcript was found overlapping with, but opposite in direction to, the ICP0 transcript (and was therefore "anti-sense"). Although this transcript has been only partially characterized, we believe that it may have a role in maintaining the latency of herpes simplex virus.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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