miRNA 146a promotes chemotherapy resistance in lung cancer cells by targeting DNA damage inducible transcript 3 (CHOP).

The underlying molecular mechanism of lung cancer drug resistance is poorly understood. The mediator of endoplasmic reticulum stress CHOP (DNA damage inducible transcript 3) promotes stress-induced apoptosis and appears to function as a transcription factor in multiple diseases. However, its potenti...

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Published in:Cancer Letters Vol. 428; pp. 55 - 69
Main Authors: Tan, Wenfeng, Liao, Yi, Qiu, Yang, Liu, Hongxiang, Tan, Deli, Wu, Tao, Tang, Meng, Zhang, Shixin, Wang, Haidong
Format: research Journal Article
Published: Elsevier B.V. Aug2018
Online Access:View this record in EBSCOhost
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      issn: 03043835
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      dt: Aug2018
      vid: 428
      pid: 1004
      pub: Elsevier B.V.
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        129734700
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        10.1016/j.canlet.2018.04.028
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        129734700
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        atl: miRNA 146a promotes chemotherapy resistance in lung cancer cells by targeting DNA damage inducible transcript 3 (CHOP).
      aug:
        au:
          Tan, Wenfeng
          Liao, Yi
          Qiu, Yang
          Liu, Hongxiang
          Tan, Deli
          Wu, Tao
          Tang, Meng
          Zhang, Shixin
          Wang, Haidong
        affil: Cardiothoracic Surgery Department, Southwest Hospital, Army Medical University (Third Military Medical University), Chongqing, China
      sug:
        subj:
          RNA Metabolism
          Proteins
          Lung Neoplasms Drug Therapy
          Drug Resistance, Neoplasm
          Lung Neoplasms Mortality
          Lung Neoplasms Pathology
          Biochemical Phenomena
          Male
          Genes Drug Effects
          Genetic Techniques
          Mice
          Lung Neoplasms
          Animal Studies
          Proteins Metabolism
          Cell Line, Tumor
          Lung Pathology
          Kaplan-Meier Estimator
          Epithelial Cells
          Middle Age
          Female
          Human
          Prognosis
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
          Middle Aged: 45-64 years
          Male
          Female
      ab: The underlying molecular mechanism of lung cancer drug resistance is poorly understood. The mediator of endoplasmic reticulum stress CHOP (DNA damage inducible transcript 3) promotes stress-induced apoptosis and appears to function as a transcription factor in multiple diseases. However, its potential contributions to multidrug resistance in solid tumors is unknown. Here, we investigated CHOP expression in tumor tissues form 69 lung cancer patients, finding that deficient CHOP expression is associated with poor prognosis. Cisplatin-resistant lung cancer cells exhibited lower expression of CHOP compared to that in sensitive lung cancer cells, and silencing or augmenting CHOP expression enhanced or impaired cisplatin resistance, respectively. Mechanistic investigations revealed that CHOP is directly associated with the regulation of autophagy or apoptosis-regulatory genes including LC3-II, death receptor 5 (DR5), and telomere repeat-binding factor 3. Notably, CHOP was identified as a target of miR-146a, and increased miR-146a expression in lung cancer cells was suggested to be responsible for CHOP mRNA down-regulation. Further, animal models confirmed that abnormally expressed miR-146a in lung cancer cells does not affect growth, but rather alters chemotherapy sensitivity. Together, CHOP is a useful prognostic marker and miR-146a is a potential therapeutic target for multidrug-resistant lung cancer.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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