Cellular Senescence Biomarker p16INK4a+ Cell Burden in Thigh Adipose is Associated With Poor Physical Function in Older Women.

Background: Ample evidence implicates cellular senescence as a contributor to frailty and functional decline in rodents, but considerable effort remains to translate these findings to human aging.Methods: We quantified senescence biomarker p16INK4a-expressing cells in thigh adipose tissue obtained f...

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Publicado en:Journals of Gerontology Series A: Biological Sciences & Medical Sciences Vol. 73; no. 7; pp. 939 - 946
Autores principales: Justice, Jamie N., Gregory, Heather, Tchkonia, Tamar, LeBrasseur, Nathan K., Kirkland, James L., Kritchevsky, Stephen B., Nicklas, Barbara J.
Formato: research Journal Article
Publicado: Oxford University Press / USA Jul2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul2018
      vid: 73
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      pub: Oxford University Press / USA
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        10.1093/gerona/glx134
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        atl: Cellular Senescence Biomarker p16INK4a+ Cell Burden in Thigh Adipose is Associated With Poor Physical Function in Older Women.
      aug:
        au:
          Justice, Jamie N.
          Gregory, Heather
          Tchkonia, Tamar
          LeBrasseur, Nathan K.
          Kirkland, James L.
          Kritchevsky, Stephen B.
          Nicklas, Barbara J.
        affil: Sticht Center for Healthy Aging and Alzheimer's Prevention, Internal Medicine - Gerontology and Geriatric Medicine, Wake Forest School of Medicine (WFSM), Winston-Salem, North Carolina
      sug:
        subj:
          Adipose Tissue Metabolism
          Aging
          Aging Physiology
          Proteins Metabolism
          Adipose Tissue Physiopathology
          Obesity Pathology
          Resistance Training
          Obesity Physiopathology
          Human
          Frail Elderly
          Adipose Tissue Pathology
          Adipose Tissue Distribution
          Female
          Obesity Metabolism
          Cross Sectional Studies
          Body Mass Index
          Thigh
          Diet Therapy
          Aged
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
          Clinical Assessment Tools
          Scales
          Funding Source
          Aged: 65+ years
          Female
      ab: Background: Ample evidence implicates cellular senescence as a contributor to frailty and functional decline in rodents, but considerable effort remains to translate these findings to human aging.Methods: We quantified senescence biomarker p16INK4a-expressing cells in thigh adipose tissue obtained from older women previously enrolled in a 5-month resistance training intervention, with or without caloric restriction (RT ± CR, n = 11 baseline, 8 pre-post-intervention pairs). Women in this subsample were older (72.9 ± 3.4 y) and overweight/obese (body mass index: 30.6 ± 2.4 kg/m2). p16INK4a+ cells were identified from 12 to 20 random visual fields/sample at 20× magnification (immunohistochemical, nuclear staining) and were present in all adipose samples.Results: Cross-sectional associations were observed between p16INK4a+ cell burden and physical function, including grip strength (r = -0.74), 400-m walk time (r = 0.74), 4-m gait speed (r = -0.73), and self-perceived mobility (r = -0.78) (p ≤ .05). These relationships remained significant after independent adjustments for age and adiposity (p ≤ .05). p16INK4a+ cell abundance was lower following the intervention (pre: 5.47 ± 3.4%, post: 2.17 ± 1.1% count p16INK4a+ cells, p ≤ .05).Conclusions: These results provide proof-of-concept that p16INK4a+ cells in thigh adipose are associated with physical function, and may be sensitive to change with RT ± CR in overweight/obese older women.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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