Suppression of TRIM72‐mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing.
Foot ulcers are amongst the most prevalent complications of diabetes, known for their delayed healing process. Recent research indicates that the transcription factor forkhead box M1 (FOXM1) plays a role in promoting diabetic ulcer repair. However, the precise mechanisms underlying FOXM1 functions i...
| Publicado en: | Wound Repair & Regeneration Vol. 33; no. 1; pp. 1 - 14 |
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| Autores principales: | , , , , , , , , |
| Formato: | pictorial research tables/charts Journal Article |
| Publicado: |
Wiley-Blackwell
Jan/Feb2025
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| Acceso en línea: | Ver este registro en EBSCOhost |