Microsatellite Instability in Sarcoma: Fact or Fiction?

Microsatellite instability (MSI) is a unique molecular abnormality, indicative of a deficient DNA mismatch repair (MMR) system. Described and characterized in the colorectal cancer literature, the MSI-positive phenotype is predictive of disease susceptibility, pathogenesis, and prognosis. The clinic...

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Publicado en:ISRN Oncology pp. 1 - 10
Autores principales: Monument, Michael J., Lessnick, Stephen L., Schiffman, Joshua D., Randall, Rl. Tx.
Formato: equations & formulas historical material pictorial review tables/charts Journal Article
Publicado: Wiley-Blackwell 2012
Acceso en línea:Ver este registro en EBSCOhost
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        atl: Microsatellite Instability in Sarcoma: Fact or Fiction?
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          Monument, Michael J.
          Lessnick, Stephen L.
          Schiffman, Joshua D.
          Randall, Rl. Tx.
        affil: Sarcoma Services, Department of Orthopaedics, Huntsman Cancer Institute, University of Utah School of Medicine, 2000 Circle of Hope, Salt Lake City, UT 84112, USA
      sug:
        subj:
          Sarcoma Physiopathology
          Genomics
          Funding Source
          History of Medicine
          Polymerase Chain Reaction Utilization
          Sarcoma, Ewing's
      ab: Microsatellite instability (MSI) is a unique molecular abnormality, indicative of a deficient DNA mismatch repair (MMR) system. Described and characterized in the colorectal cancer literature, the MSI-positive phenotype is predictive of disease susceptibility, pathogenesis, and prognosis. The clinical relevance of MSI in colorectal cancer has inspired similar inquisition within the sarcoma literature, although unfortunately, with very heterogeneous results. Evolving detection techniques, ill-defined sarcoma-specific microsatellite loci and small study numbers have hampered succinct conclusions. The literature does suggest that MSI in sarcoma is observed at a frequency similar to that of sporadic colorectal cancers, although there is little evidence to suggest that MSI-positive tumors share distinct biological attributes. Emerging evidence in Ewing sarcoma has demonstrated an intriguing mechanistic role of microsatellite DNA in the activation of key EWS/FLI-target genes. These findings provide an alternative perspective to the biological implications of microsatellite instability in sarcoma and warrant further investigation using sophisticated detection techniques, sensitive microsatellite loci, and appropriately powered study designs.
      pubtype: Academic Journal
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        equations & formulas
        historical material
        pictorial
        review
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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