Engineering the gut microbiota to treat hyperammonemia.

Increasing evidence indicates that the gut microbiota can be altered to ameliorate or prevent disease states, and engineering the gut microbiota to therapeutically modulate host metabolism is an emerging goal of microbiome research. In the intestine, bacterial urease converts host-derived urea to am...

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Publicado en:Journal of Clinical Investigation Vol. 125; no. 7; pp. 2841 - 2851
Autores principales: Shen, Ting-Chin David, Albenberg, Lindsey, Bittinger, Kyle, Chehoud, Christel, Chen, Ying-Yu, Judge, Colleen A, Chau, Lillian, Ni, Josephine, Sheng, Michael, Lin, Andrew, Wilkins, Benjamin J, Buza, Elizabeth L, Lewis, James D, Daikhin, Yevgeny, Nissim, Ilana, Yudkoff, Marc, Bushman, Frederic D, Wu, Gary D
Formato: Journal Article
Publicado: American Society for Clinical Investigation Jul2015
Acceso en línea:Ver este registro en EBSCOhost
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        atl: Engineering the gut microbiota to treat hyperammonemia.
      aug:
        au:
          Shen, Ting-Chin David
          Albenberg, Lindsey
          Bittinger, Kyle
          Chehoud, Christel
          Chen, Ying-Yu
          Judge, Colleen A
          Chau, Lillian
          Ni, Josephine
          Sheng, Michael
          Lin, Andrew
          Wilkins, Benjamin J
          Buza, Elizabeth L
          Lewis, James D
          Daikhin, Yevgeny
          Nissim, Ilana
          Yudkoff, Marc
          Bushman, Frederic D
          Wu, Gary D
      sug:
        subj: Microbiota
      ab: Increasing evidence indicates that the gut microbiota can be altered to ameliorate or prevent disease states, and engineering the gut microbiota to therapeutically modulate host metabolism is an emerging goal of microbiome research. In the intestine, bacterial urease converts host-derived urea to ammonia and carbon dioxide, contributing to hyperammonemia-associated neurotoxicity and encephalopathy in patients with liver disease. Here, we engineered murine gut microbiota to reduce urease activity. Animals were depleted of their preexisting gut microbiota and then inoculated with altered Schaedler flora (ASF), a defined consortium of 8 bacteria with minimal urease gene content. This protocol resulted in establishment of a persistent new community that promoted a long-term reduction in fecal urease activity and ammonia production. Moreover, in a murine model of hepatic injury, ASF transplantation was associated with decreased morbidity and mortality. These results provide proof of concept that inoculation of a prepared host with a defined gut microbiota can lead to durable metabolic changes with therapeutic utility.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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