Visualization of tumor heterogeneity by in situ padlock probe technology in colorectal cancer.

Tumor heterogeneity is considered a major cause for therapy resistance in colorectal cancer. Sub-populations of cells with different genetic alterations may exist in spatially distinct areas. Upon therapy, resistant sub-clones may enrich and ultimately lead to disease progression. Although ample dat...

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Publicado en:Histochemistry & Cell Biology Vol. 148; no. 2; pp. 105 - 116
Autores principales: El-Heliebi, Amin, Kashofer, Karl, Fuchs, Julia, Jahn, Stephan, Viertler, Christian, Matak, Andrija, Sedlmayr, Peter, Hoefler, Gerald
Formato: Journal Article
Publicado: Springer Nature Aug2017
Acceso en línea:Ver este registro en EBSCOhost
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      pub: Springer Nature
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        atl: Visualization of tumor heterogeneity by in situ padlock probe technology in colorectal cancer.
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          El-Heliebi, Amin
          Kashofer, Karl
          Fuchs, Julia
          Jahn, Stephan
          Viertler, Christian
          Matak, Andrija
          Sedlmayr, Peter
          Hoefler, Gerald
        affil: Institute of Cell Biology, Histology and Embryology , Medical University of Graz , Harrachgasse 21/VII 8010 Graz Austria
      sug:
      ab: Tumor heterogeneity is considered a major cause for therapy resistance in colorectal cancer. Sub-populations of cells with different genetic alterations may exist in spatially distinct areas. Upon therapy, resistant sub-clones may enrich and ultimately lead to disease progression. Although ample data are available on tumors which are heterogeneous on a morphological level, only little is known about morphologically homogeneous tumors. We aimed to investigate if morphologically homogeneous colorectal cancer can harbor a heterogeneous genetic landscape. We chose to microdissect six morphologically homogeneous colorectal carcinomas into several areas and performed next-generation sequencing (NGS) to identify tumors with genetic heterogeneity. We then applied an mRNA-based in situ mutation detection technology based on padlock probes to localize and visualize mutations directly in the tumor tissue. In three out of six tumors, NGS revealed a high rate of variability of mutations between different tumor areas. We selected two cases for in situ mutation detection to visualize genetic heterogeneity. In situ mutation detection confirmed differences in mutant allele frequencies between different tumor areas of morphological homogeneous tumors. We conclude that genetic heterogeneity in morphologically homogeneous colorectal cancer is an observable, but underreported event. Our results illustrate the power of in situ mutation analysis to visualize genetic heterogeneity directly in tumor tissue.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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